Recognition of Plant Poisoning
Call a Veterinarian
How to Prevent Plant Poisoning
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Scouring Rush (Equisetum Hyemale)
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Sorghum. Sorghum vulgare L.
Sudan grass. Sorghum vulgare var. sudanese (Piper) Hitch.
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fff00006.gif Star-of-Bethlehem (Ornithogalum
umbellatum L.) Plate 6.
fff00007.gif Pokeweed, pokeberry, poke. Phytolacca
americana L. Plate 7.
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fff00011.gif fff00012.gif Horsechestnut. Aesculus Hippocastanum
L. Plate 10. Fig. 3.
fff00013.gif St. Johnswort, Klamath weed,
rosin-rose. Hypericum perforatum L. Plate 13.
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fff00015.gif Mountain laurel, calico-bush,
poison laurel. Kalmia latifolia L. Plate 15.
fff00016.gif Indian hemp, dogbane. Apocynum
cannabinum L. Plate 16. fff00017.gif Swamp milkweed. Asclepias incarnata
L. Plate 17. Fig. 4.
fff00018.gif Jimson weed, thorn-apple, devil's
trumpet. Datura stramonium L. Plate 18.
fff00019.gif Black nightshade, deadly nightshade,
garden nightshade, poison berry. Solonum nigrum L. Plate 19.
fff00020.gif Indian tobacco. Lobelia inflata
L. Plate 20. Fig. 1.
fff00021.gif fff00022.gif Sneezeweed, staggerwort, swamp
sunflower. Helenium autumnale L. Plate 22. Fig. 1.
fff00023.gif Cocklebur, clotbur, sheep-bur.
Xanthium orientale L. (X. commune Britt.) Plate 23. fff00024.gif Kentucky coffee tree. Gymnocladus
dioica L. Koch. Plate 24.
Supplementary List of Poisonous Plants
Ergot (Claviceps purpurea (Fr.) Tul.)
Jack-in-the-pulpit, Indian turnip (Arisaema atrorubens (Ait.) Blume)
Hemp, marijuana (Cannabis sativa L.)
Buttercup (Ranunculus spp. )
Moonseed (Menisperum canadense L.)
White sweet clover (Melilotus alba Desr.)
Alsike clover (Trifolium hybridum L.)
Spurges (Euphorbia spp. )
Castor bean, castor oil plant (Ricinus communis L.)
Hogwort, wooly croton (Croton capitatus Michx.)
Boxwood (Buxus sempervirens L.)
Climbing bittersweet (Celastrus scandens L.)
Ground ivy, Gill-over-the-ground, Creeping Charlie (Glechoma hederacea
L.)
Blue nightshade, European nightshade (Solanum dulcamara L.)
Horse nettle, bull nettle (Solanum carolinense L.)
Tobacco (Nicotiana Tabacum L.)
Matrimony vine (Lycium halimifolium Mill.)
Buttonbush (Cephalanthus occidentalis L.)
Elderberry, black eider (Sambucus canadensis L.)
Ragwort, groundsel (Senecio spp. )
Plants Causing Mechanical Injury
Glossary
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Suggestions for Sending Plant Specimens for Identification
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Horsetail. Equisetum arvense L. Plate 1. Fig. 1.
Scouring rush. Equisetum hyemale L. Plate 1. Fig. 2.
DESCRIPTION. Shoots round, hollow, jointed, of two types, arising
from a perennial, creeping, underground rootstock. One type of shoot, tan
in color, appears in early spring, and bears the reproductive spores in
a terminal, cone-like structure. The other is sterile and appears later,
bearing whorls of pine-needle-like branches. Leaves reduced to "teeth"
and arranged in whorls around the stem at the joint.
CONDITIONS OF POISONING. These plants contain, among other toxic
principles, aconitic acid, equistitine, and thiaminase. Horses are much
more susceptible to Equisetum than cattle.
SYMPTOMS. Symptoms appear suddenly after the plant has been
eaten, especially if the plant was in a dried condition. The first symptoms
are unthriftiness, weakness, with loss of flesh. Appetite may or may not
be affected. In a few weeks a lack of muscle control will be noticeable,
and the animal will frequently fall. In advanced cases the symptoms are
difficult breathing, pale mucous membranes, rapid and weak pulse, diarrhea,
convulsion, coma, and then death.
TREATMENT. In early stages purgatives and stimulants should
be given. The animal should be kept quiet, and high quality rations should
be fed. Avoid feeding hay containing large quantities of the plant. Treatment
with massive doses of thiamine seems to be effective except in advanced
cases.
DESCRIPTION. Shoots similar, forming long, tapering canelike
stalks, 1 to 6 feet high. Stems stiff, evergreen, with terminal spore-bearing
cones. Leaves reduced to "teeth" and arranged in whorls around the stem
at the joints. Common in wet localities, thickets, along streams, roadside
ditches and borders of swamps, and in mountain sections of the state.
CONDITIONS OF POISONING. Same as Equisetum arvense L.
SYMPTOMS. Same as Equisetum arvense L.
TREATMENT. Same as Equisetum arvense L.
Brake fern or Bracken. Pteridium latiusculum Mason. (Pteris aquilina
L.) Plate 2.
DESCRIPTION. Coarse-growing perennial fern, with stout black
horizontal rootstock. Frond or leaf broad, triangular in shape, and divided
into three main parts, each of which is twice subdivided. Reproduction
by spores borne in late summer on the lower edges of the mature fronds.
CONDITIONS OF POISONING. Not ordinarily eaten by stock, except
during a dry season or in late summer when there is a scarcity of green
herbage. Poisonous both fresh and dried in hay, though hogs eat the rootstocks
without harm. The chemical action causing the poison is cumulative and
symptoms often do not appear immediately. The toxic material is unknown.
SYMPTOMS. Cattle affected usually have a high temperature, stand
with head down and drool at the mouth. There is a rapid loss of flesh along
with difficult breathing and excess salivation. Blood appears in the feces,
either in bright red clots or as black feces. There is generally a trickle
of blood from the nostrils, The mucous membranes will be congested, hemorrhagic
or a yellow color. These symptoms may be confused with anthrax.
TREATMENT. If the condition is diagnosed early enough, some
animals will respond to treatment. Saline purgatives, raw linseed oil,
mineral oil or melted lard should be given. Thiamine hydrochloride injected
intravenously may be of some value.
Yew. Taxus spp.
DESCRIPTION. Evergreen shrub. Leaves alternate, needle-like,
stiff, 1/2 to 1 inch long, dark green and glossy above and yellow-green
on the lower surface. Seeds solitary, born in a cup-shaped, fleshy bright
red fruit, appearing somewhat like a red berry.
CONDITIONS OF POISONING. Poisonous properties have been attributed
to the alkaloid, taxine, a heart depressant. The leaves, bark, wood and
seeds are poisonous. When large amounts are eaten, death may result within
a short time. Cattle, horses, sheep, goats, and wild animals have been
poisoned by browsing the leaves and twigs. The red pulp of the fruits seems
to be harmless, but seeds may contain the poisonous alkaloid. Poisoning
sometimes occurs from livestock eating prunings and droppings. The red
fruits are attractive to children.
SYMPTOMS. Large doses usually result in sudden death without
additional symptoms. Animals are found close to plants from which they
have eaten, frequently with twigs or leaves still in their mouths. In subacute
cases, typical symptoms include trembling, labored breathing, and collapse.
Spontaneous recovery in subacute cases has been reported, and, in such
cases, the time between ingestion and evidence of symptoms may be as long
as two days. Gastroenteritis and diarrhea may be evidenced in subacute
cases.
TREATMENT. In acute cases, death occurs before any treatment
can be administered. In less acute cases, atropine has been reported as
an effective treatment to counteract the depressant effect of the alkaloid
taxine.
Sorghum. Sorghum vulgare L.
Johnson grass. Sorghum halpense L. Pers. Plate 4.
Sudan grass. Sorghum vulgare var. sudanense (Piper) Hitch.
Johnson grass. Sorghum halpense L. Pers.
DESCRIPTION. Large perennial, 3-6 feet high, with scaly, creeping
rootstalks. Flowers in long, open, terminal panicle.
DESCRIPTION.--Very sturdy annual. Tall, coarse grass with flowers
similar to Johnson grass.
DESCRIPTION. Annual, similar to sorghum, but of more slender
growth. Neither sorghum nor sudan grass has scaly, creeping rootstalks.
CONDITIONS OF POISONING. Normally these grasses furnish excellent
forage, and their use should not be discouraged. Since, under certain conditions,
they may be dangerously poisonous, they should never be fed in a wilted
or stunted condition to livestock. Cut hay and silage should be cured for
at least six weeks before being used. These grasses should never be pastured
until they are at least a foot tall; second growth is also very dangerous.
SYMPTOMS. LethaI amounts of hydrocyanic acid cause respiratory
paralysis and spasms, followed quickly by death. Animals are usually found
dead before symptoms are noticed.
TREATMENT. If treatment is to be beneficial it should be started
before respiratory paralysis begins to occur.
Oaks. Quercus spp. L. Plate 5.
CONDITIONS OF POISONING.-Oaks seem to be most poisonous in the
budding and leafing stages, which occur early in the spring when very little
other forage is available. Animals turned into wooded areas before grass
becomes abundant may consume large quantities of the buds and young leaves.
Small amounts do not seem to be injurious, but when they are a large part
of an animal's diet, severe illness can follow, often resulting in death
of the animal two weeks to a month later. Cattle and sheep seem more susceptible
to oak poisoning than other animals.
SYMPTOMS. Constipation and loss of appetite are early symptoms.
The feces are scanty, dry, and often contain mucus and blood. In some cases
this may later be followed by profuse diarrhea. The affected animal usually
exhibits a gaunt, tucked up appearance. There is evidence of jaundice,
and blood or hemoglobin may appear in the urine. The muzzle may become
dry and scaly. There is a reluctance on the part of the affected animal
to follow the herd and a tendency to remain near water. When forced to
move, severely affected animals show evidence of weakness and have a shuffling
gait. They later collapse and die. Rarely are more than a few animals in
a herd affected.
TREATMENT. Animals grazing wooded areas in the spring should
be watched for early evidence of oak poisoning. If it occurs, animals should
be moved to other pastures. In early or mild cases of oak poisoning, veterinarians
may administer symptomatic treatment with good success. However, for severely
poisoned animals no effective treatment is known.
DESCRIPTION. Star-of-Bethlehem is a perennial that reproduces
mostly by bulbs, rarely by seed. It belongs to the same family as wild
garlic (lily family) and may be confused with wild garlic. Star-of-Bethlehem
does not, however, have the strong scent of onion or garlic, and the leaves
are about as long as the stem and have a light green midrib. The stems
develop from small bulbs that are usually in clumps, grow to a height of
from 4 to 12 inches, and bear several white star-shaped flowers.
CONDITIONS OF POISONING. All parts of the plant are poisonous
when eaten either fresh or in hay. It is reported to contain uncharacterized
alkaloids. The bulb seems to be more toxic than other parts of the plant.
SYMPTOMS. Symptoms include depression, salivation, and bloating.
TREATMENT. No effective treatment known.
DESCRIPTION. TalI, smooth, perennial herb with a large taproot.
Stems succulent, purplish, 3 to 10 feet high. Leaves alternate, pointed,
narrowly oblong, with smooth margins. Flowers small, white, borne in long,
drooping clusters. Fruits flattened, spherical, dark purple berries, usually
with ten seeds.
CONDITIONS OF POISONING. A saponin-like substance, in addition
to the alkaloid, phytolaccine, is present in all parts of the plant, especially
the roots and seeds. Cooking destroys the poison and young shoots are sometimes
used as cooked greens. Although animals usually avoid pokeweed due to its
unpleasant taste, early in the spring they sometimes feed on the young
shoots. Cases have been reported of poisoning in pigs from rooting out
and consuming the roots of the plant. It has also been reported that in
Kentucky cattle were attracted to pokeweed plant treated with certain herbicides,
thus causing loss of several animals.
SYMPTOMS. In most cases when small amounts are eaten, the only
symptoms are retching and vomiting, usually noticed several hours after
the plant has been eaten. If larger amounts are eaten, spasms, diarrhea,
and convulsions will be the main symptoms. The cause of death is respiratory
paralysis.
TREATMENT. Tannic acid, mineral oil and stimulants will help
in most cases if the condition is diagnosed early enough.
Dwarf larkspur, staggerweed. Delphinium tricorne Michx. Plate 8.
DESCRIPTION. Stout perennial, 4-35 inches high. Leaves alternate
and very deeply lobed. Flowers spurred, blue or occasionally white, arranged
in terminal clusters, appearing in spring. The root is a tuberous cluster.
CONDITIONS OF POISONING. Larkspur contains several very poisonous
alkaloids, including delphinine, and is most poisonous in the early stages
of growth, during April and May. Poisoning occurs when stock grazes in
woodland pastures before other green herbage is available. Cattle are the
most susceptible, but horses and sheep can be poisoned by eating large
quantities.
SYMPTOMS. The symptoms of larkspur poisoning differ according
to the amount eaten and the animal's tolerance. Small amounts may cause
loss of appetite; excitability, staggering, and constipation. Severe symptoms
that develop when an animal eats large quantities are slobbering, vomiting,
colic, bloating, and convulsions. Death is due to respiratory paralysis.
TREATMENT. Protect animals from excitement by keeping them in
a quiet place and give them such drugs as chloral hydrate or one of the
barbiturates. Epsom salts may be given to help the constipation. Other
drugs should be given to relieve the animal. It may be necessary to treat
the animal for bloat.
Dutchman's breeches, staggerweed, wild bleeding heart. Dicentra Cucullaria
L. Bernh. Plate 7. Fig. 1.
Squirrel corn, staggerweed, wild bleeding heart. Dicentra canadensis.
(Goldie) Walp. Plate 9. Fig. 2.
DESCRIPTION. Delicate perennial with finely cut fern-like leaves.
Small yellow pea-like tubers arranged along the underground stem. Flowers
1 to 10, creamy white with 2 short rounded projections, and arranged on
slender stalks.
DESCRIPTION. Closely resembles squirrel corn but small grain-like
tubers are clustered at the base of the stem. Flowers with 2 spur-like
projections.
CONDITIONS OF POISONING. The entire plants of both species contain
a number of poisonous alkaloids. Dutchman's breeches is more poisonous
than squirrel corn. These plants are unpalatable and are not frequently
eaten in harmful quantities when other forage is available. Most cases
of poisoning occur in early spring (April or May) when animals are grazing
in wooded areas. Cows are more frequently poisoned than horses; sheep are
not affected.
SYMPTOMS. A staggering gait and a loss of milk production. Later
symptoms are sudden trembling which increases in severity, frothing of
the mouth, labored breathing, diarrhea and convulsions. Most animals will
recover, if the dosage is not too heavy, and if they are kept away from
the plants after the first symptoms appear.
TREATMENT. Animals showing "staggers" in the spring should be
moved to clean pastures at once. If poisoning symptoms are severe, purgatives,
mineral oil and stimulants may be given.
Wild black cherry. Prunus serotina Ehrh. Plate 10.
DESCRIPTION. Tree or shrub with slender horizontal branches.
Bark of young branches and twigs reddish-brown with prominent white lenticels
(pores). Leaves alternate, simple, elliptical, pointed, margins finely
toothed, leathery in texture, and usually have a row of hairs on the lower
surface along both sides of the midrib. Flowers small, white, in drooping
clusters, and produce dark-red to black cherry fruits.
CONDITIONS OF POISONING. Wild cherries contain the glucoside
amygdalin, which, by a series of chemical changes, is converted to hydrocyanic
acid. This acid is formed very rapidly in wilted or bruised leaves, particularly
those on young tender shoots. Most cases of poisoning occur when animals
have access to wilted leaves on branches blown down during wind or hail
storms, or branches that have been clipped or pruned.
SYMPTOMS. Symptoms may develop very rapidly after an animal
has eaten wilted wild cherry leaves. The poisoned animal becomes uneasy,
staggers, and has convulsions. Breathing will be very difficult and the
mucous membranes become blue. Death may come so quickly that the animal
will be found near the wilted leaves or tree.
DESCRIPTION. Moderate-sized tree, often with 2 short spines
at base of leafstalk, bark rough. Leaves alternate, pinnately compound;
the individual leaflets oval-shaped, without teeth. Flowers creamy white,
fragrant, sweet-pea like, arranged in long, drooping,.clusters. Fruit a
flat, brown pod, 1/2 inch wide, 2 to 4 inches long, and containing 4-8
small kidney-shaped beans.
CONDITIONS OF POISONING. The poisonous substance is a phytotoxin,
robin. Animals are affected by eating the young shoots, leaves, pods, seeds,
and by gnawing on the bark, or drinking water in which the pods have been
soaked. All farm animals are susceptible.
SYMPTOMS. Animals will become stupid, not notice their surroundings,
and stand with the legs apart. Heart action is irregular and breathing
is feeble, mucous membranes are yellow, and the pupil of the eye dilated.
Colic pains may be present and soon followed by diarrhea. Cattle are quite
often dizzy and very nervous.
TREATMENT. Death follows the onset of symptoms unless treatment
is started soon. An injection of digitalis to help the heart action is
useful. Other treatments used are just to help decrease the symptoms and
give ease to the animal.
Sweet buckeye. Aesculus octandra Marsh. Plate 10. Fig. 2.
Ohio buckeye. Aesculus glabra Willd. Plate 12. Fig. 1.
DESCRIPTION. Tree or shrub. Leaves opposite, palmately compound
(leaflets arranged like the fingers of a hand). Leaflets usually five.
Flowers yellowish, in large clusters at ends of branches. Fruit a prickly
capsule (at least when young), leathery, 1 to 3 seeded-each seed glossy
brown with a pale scar-hence the common name buckeye. Bark with a strong,
offensive odor. Common in rich, moist woods, and along river banks.
DESCRIPTION. Similar to Ohio buckeye, but leaflets usually seven
and flowers white.
DESCRIPTION. Similar to Ohio buckeye, but bark with a slight
odor, and fruits not prickly. Common in rich, moist woods and along river
banks.
CONDITIONS OF POISONING. These plants contain poisonous alkaloids
and glucosides, especially in the young shoots, nuts, and sprouts. Poisoning
is most likely to occur in early spring in woodland pastures where there
are sprouts and seedlings.
SYMPTOMS. Buckeye poisoning affects the central nervous system,
causing an uneasy, staggering gait. The animal will be weak, have severe
trembling, and may vomit. Mucous membranes will be congested and the pupils
of the eye will be dilated. The animal then will go into a coma before
death.
TREATMENT. Quite often the animal is found dead before any treatment
can be given. If cases are found soon. enough, stimulants and purgatives
are indicated.
DESCRIPTION. Erect, freely branching perennial, 1 to 3 feet
tall. Leaves opposite, less than 1/2 inch wide, oblong or linear in shape,
with smooth margins, and covered with transparent dots. Many yellow flowers
in somewhat flattened clusters at the top of the,,stem. Petals with black,
glandular dots on the margins. The fruit is a three-parted capsule containing
many small, dark-brown seeds.
CONDITIONS OF POISONING. The poisonous action of this plant
is probably due to two fluorescent substances, hypericin and hypericum
red. When white-skinned animals feed on large quantities of St. Johnswort
in the flowering stage, their skins may become extremely sensitive to sunlight,
a condition called photosensitization. Dark-skinned animals have enough
pigment to screen out the undesirable light rays.
SYMPTOMS. White-skinned animals, animals with white spots or
belts, or sheep that have been sheared, quite often develop a dermatitis,
the result of light sensitization. The most typical symptoms are skin blisters,
falling hair and scabs. The skin may become dry and crack open. Poisoning
is rarely fatal. In some cases in sheep, paralysis has been noted.
TREATMENT. Remove animals from the pasture as soon as possible
and put them in a cool, dark barn. Apply oil to the affected areas. Supply
plenty of fresh water and feed.
Poison hemlock, spotted hemlock, deadly hemlock. Conium maculatum L.
Plate 14
DESCRIPTION. Smooth biennial herb, 3 to 8 feet high, with purple-spotted
stem and finely-divided compound leaves. Flowers small, white, in umbels,
blossom in early summer. Leaves have a rank, disagreeable odor. Poison
hemlock can be distinguished from 'Water hemlock by more finely-divided
leaves and a long, parsnip-like root.
CONDITIONS OF POISONING. Poisoning is most likely to occur in
early spring when leaves are green and other pasturage is not available.
The entire plant is poisonous. Cases of human poisoning have occurred from
mistakenly eating the seeds, leaves, and roots for anise, parsley or parsnips.
The toxic principle is the volatile alkaloid, coniine. Although drying
reduces the coniine content, hay containing the dried plants is not entirely
safe.
SYMPTOMS. Poisoning generally appears suddenly, and the owner
finds the animal "down." Some of the symptoms that may be noticed are:
excessive salivation (slobbering), loss of appetite, muscular weakness
or twitching of the muscles, incoordination, rapid pulse and great pain.
Death occurs from respiratory paralysis.
TREATMENT. If animals are found and diagnosed early enough,
purgatives may be given to empty the digestive tract. Intestinal astringents
such as tannic acid are useful. Nerve and heart stimulants may be given.
DESCRIPTION. Evergreen shrub, 3-9 feet high. Leaves to five
inches long, alternate or irregular, leathery, shiny, light green on lower
surface. Flowers pink or white, cup-shaped, borne in spring. Common in
upland woods, hilly pastures, and on acid soil.
DESCRIPTION. Evergreen shrub or small tree. Leaves alternate,
leathery, whitish or rusty on lower surfaces. Flowers bell-shaped, rose-pink
to white. Common in damp woods, swamps, and upland areas.
CONDITIONS OF POISONING. Mountain laurel and rhododendron contain
a resinous substance, andromedotoxin. Sheep are more frequently poisoned
than other livestock, since they are often pastured in areas that are better
adapted for the growth of these plants.
SYMPTOMS. In certain sections of the state, quite a few animals
are lost each year from eating laurel and rhododendron. If an animal eats
0.2% of its body weight, symptoms may develop. The first symptom is irregular
breathing. Later, slobbering at the mouth, grating of the teeth, vomiting,
staggering, blindness, stupor, and then death. The meat from these animals
should not be eaten. If the animal recovers, several days should elapse
before it is used for human consumption.
TREATMENT. If the animals are found early enough, oil drenches,
with materials such as mineral oil, raw linseed oil, lard, etc. should
be given.
DESCRIPTION. Branching perennial, 1 1/2 to 5 feet high. Stem
contains a milky juice or latex and arises from a vertical underground
rootstock. Leaves opposite, oblong in shape, with smooth margins. Flowers
greenish-white, borne in clusters at the ends of the stems and branches.
Fruit a long, slender pod containing many seeds that bear tufts of "floss."
CONDITIONS OF POISONING. Indian hemp contains the glucoside
cymarin, poisonous resins and possibly other toxic substances. All parts
of the plant are poisonous, either fresh or dried in hay. As little as
1/2 to 1 ounce of the fresh green leaves can cause death to a cow.
SYMPTOMS. A rise in body temperature, sweating, strong pulse,
ears and legs become cold, pupils of the eyes usually dilated, and the
inside of the mouth becomes red and sore. Bowel action is frequent. In
advanced cases death will usually occur.
TREATMENT. If cases are diagnosed early enough, the stomach
should be emptied and gallic or tannic acid given as an antidote. Heart
stimulants are sometimes helpful. Keep the animal in a quiet place and
give it good feed and water.
Common milkweed. Asclepias syriaca L. Plate 17. Fig. 1.
Butterfly weed. Asclepias tuberosa L. Plate 17. Fig. 2
Whorled milkweed. Asclepias verticillata L. Plate 17. Fig. 3.
DESCRIPTION. Stems solitary or clustered, 1-5 feet high. Leaves
opposite. Flowers pink to rose-purple, arranged in umbels. Fruit a pod
containing seeds tufted with "floss."
DESCRIPTION. Erect perennial, 2-4 feet high, stem with broad
opposite or whorled leaves and milky sap. Flowers dull pink, arranged in
simple umbels. Fruit a pod containing seeds tufted with "floss."
DESCRIPTION. Erect perennial 1-3 feet high, stems with alternate
leaves and no milky sap. Flowers bright orange, arranged in simple umbels.
Fruit a pod containing seeds tufted with "floss."
DESCRIPTION. SIender perennial, 1-3 feet high. Leaves arranged
in whorls, very narrow with curled margins. Flowers greenish-white, in
small umbels. Fruit a pod containing seeds tufted with "floss."
SYMPTOMS. The first symptoms are loss of appetite and diarrhea.
Later, the animal will stagger, fall and develop paralysis of the rear
limbs. Breathing will be labored and spasms violent. Death is due to respiratory
failure. Autopsies show congestion of liver, kidneys, heart, lungs and
nervous system.
TREATMENT. Sedatives, such as chloral hydrate and the barbiturates
are helpful in easing the pain. Mineral oil will be helpful in early stages.
DESCRIPTION. A stout, coarse annual, 2 to 5 feet tall, with
spreading branches. Leaves alternate, coarsely toothed, green or purplish
and strongly scented. Flowers trumpet shaped, large, white or purplish.
Fruit a hard, spiny capsule that splits into 4 valves at maturity.
CONDITIONS OF POISONING. Jimson weed contains the poisonous
alkaloids, hyoscyamine and hyoscine. The entire plant is poisonous, both
green and dried. Because of its strong odor and bitter taste, the green
plant is rarely eaten. Humans may be poisoned by eating the fruits.
SYMPTOMS. Common symptoms in cattle are rapid pulse and respiration,
dry mouth and, possibly, a complete retention of urine, or frequent urination.
Diarrhea, dilation of the pupils of the eyes, and stiffness may occur.
Close to death, the respirations become slow, irregular, and weak. Death
is caused by asphyxia, Quite often this weed grows in the hog lot and may
be the cause of some deaths. The main symptom in hogs is convulsive twitching
of the entire body.
TREATMENT. If animals are found early enough, mineral oil is
helpful, since it acts as a purge and coats the walls of the intestines.
Tannic acid is used to precipitate the alkaloids. Stimulants and molasses
are helpful.
DESCRIPTION. Low, branched annual, 1 to 2 feet high, with angular
stems. Leaves alternate, oval, thin textured, with wavy margins. Flowers
white, in drooping clusters on lateral stalks between the leaves, somewhat
resembling tomato flowers. Fruit a berry, green when immature, turning
purplish-black at maturity. A common weed in open woods, fields, waste
places, and around farm buildings.
CONDITIONS OF POISONING. The alkaloidal glucoside, solanine,
is present in the leaves, stems and green berries. The plants are less
toxic when dried. Most cases of poisoning occur among sheep, goats, calves,
pigs and poultry, as mature horses and cows rarely eat enough to be seriously
affected.
SYMPTOMS. The first symptoms are weakness, stupor, staggering
gait and constipation, followed by dilated pupils, loss of muscular coordination
and sense of feeling. In more advanced cases, cramps and convulsions are
typical. The animal will soon die of respiratory paralysis. The progress
of the symptoms is often rapid.
TREATMENT. There is no treatment for nightshade poisoning. Sedatives
will help stop the convulsions.
Great lobelia. Lobelia siphilitica L. Plate 20. Fig. 2
DESCRIPTION. Hairy annual with leafy branched stem, 1 to 2 feet
high. Leaves thin, oval or oblong, with toothed margins. Flowers two-lipped,
pale-blue, inconspicuous, borne in the axils of the upper leaves. Fruit
a capsule covered by the swollen inflated calyx. Common in meadows, pastures
and cultivated fields.
DESCRIPTION. PerenniaI herb to a height of 3 feet. Stem leafy,
rather stout, and usually unbranched. Flowers about 3/4 inch long, deep
blue, in a dense terminal spike.
CONDITIONS OF POISONING. Poisonous alkaloids and volatile oils
are present in the leaves, stems and fruits. In heavily infested pastures
or during dry seasons, when other green forage is scarce, animals occasionally
eat the plants in sufficient quantities to be seriously poisoned.
SYMPTOMS. Nausea, vomiting and dilated pupils. The animal will
stagger, get down, have convulsions, go into a coma and then die.
TREATMENT. Gallic or tannic acid is the best treatment, along
with stimulants. Mineral oil is helpful in lining the digestive system.
DESCRIPTION. A smooth, erect, perennial herb, 1 to 5 feet high.
Leaves opposite, oval, with pointed tips and sharply-toothed edges, strongly
resembling the leaves of nettles. The upper surface of the leaf dull, and
the lower shiny. Each leaf has 3 prominent main veins on the underside.
In late summer the small white flowers appear in compound clusters. Found
in woods; damp, shady pastures; and, occasionally, thickets and clearings.
CONDITIONS OF POISONING. Tremetol, a poisonous alcohol, is present
in the leaves and stems. This toxic principle may be transmitted through
the milk of poisoned cows to humans, causing the disease known as "milk
sickness" or "trembles." Animals are usually poisoned during the late summer
when forage is scarce. The effect is cumulative. Animals may die from eating
a large amount of the plant at one time, or from eating small amounts over
a long period of time.
SYMPTOMS. Cows appear listless, have severe constipation, violent
trembling, and their breath will become foul with a peculiar acetone odor.
Joints become stiff, the animal falls and refuses to rise or otherwise
exert itself, grinds its teeth, has rapid, labored breathing, and may die.
TREATMENT. Since poisoning usually occurs in late summer, cattle
should be removed about the first of July from areas infested with this
weed.
Bitterweed, narrow-leaved sneezeweed, yellow dog-fennel. Helenium tenuifolium
Nutt. Plate 22. Fig. 2.
DESCRIPTION. Erect-growing coarse perennial, 2 to 6 feet tall,
with narrowly-winged stem. Leaves alternate, lance-shaped and coarsely
toothed. Flowers yellow, resembling small sunflowers. Toothed ray flowers
are characteristically turned downwards. Another species, the purple-headed
sneezeweed (Helenium nudiflorum Nutt:) is less present in the state.
DESCRIPTION. Annual, 1-2 feet high, with fine, narrow leaves.
Flowers similar to sneezeweed.
CONDITIONS OF POISONING. Most cases of serious poisoning occur
in the late summer and early fall when the plants come into bloom. Sheep,
cattle and horses are susceptible. Most livestock avoid these plants, but
individual animals may eat sufficient quantities to cause death. The plants
are poisonous either fresh or cured in hay. Cows grazing on these plants
may produce milk with a bitter flavor.
SYMPTOMS. Rapid pulse, restlessness, difficult breathing, and
loss of muscular control followed by plunging and staggering blindly. At
this time the animals are extremely sensitive to the touch. After eating
large quantities of the blossoms, the animals may die suddenly with spasms
and convulsions.
TREATMENT. If the milk is bitter or the animal is showing symptoms
of poisoning, remove all the stock to clean, weed-free pastures. Melted
lard or mineral oil is helpful if given early enough.
DESCRIPTION. Branching annual, 1 to 3 feet high. Stems angled,
sometimes red-spotted. Leaves alternate, hairy, rough in texture, somewhat
heart-shaped, and variously toothed and lobed. Flowers inconspicuous, of
two kinds-those in terminal spikes produce only pollen, while those in
clusters in the axils of the leaves produce the seed. Fruit a hard, oval,
prickly bur, about 3/4 inch long, containing two seeds. The seedlings have
small strap-shaped leaves, 1/4 inch wide and 11/4 inches long, and later
produce the more characteristic leaves. They sprout from the buried burs.
Several species of cocklebur are common in Kentucky. They differ in the
shape and hairiness of the bur, but all are poisonous in the seedling stage.
CONDITIONS OF POISONING. The poisonous substance is a glucoside,
xanthostrumarin, found in germinating seeds and young seedlings. Since
the toxicity decreases as the plants develop, the mature plant is probably
non-poisonous. Hogs are most frequently poisoned, but cattle and sheep
are also susceptible. Mature plants are seldom eaten, but the ripe spiny
burs may result in purely mechanical injury.
SYMPTOMS. Depression, often accompanied by nausea and, occasionally,
vomiting. The affected animal becomes gaunt, weak, and unable to stand,
has labored respiration and a rapid, weak pulse. It may lie on its side
and move its legs in a running fashion until completely exhausted. Within
24 hours after eating 0.75% of its own weight of young seedling plants,
the animal may show signs of poisoning, and even die. If death does not
occur, several weeks will pass before the animal is back to normal.
TREATMENT. Give emergency treatment with fatty substances, such
as mineral oil, cream or even whole milk, by mouth or through a stomach
tube. Keep the animal quiet and warm.
DESCRIPTION. A large tree with rough bark, ranging from 60 to
80 feet in height. The trunk, which is relatively short and 1 to 2 feet
in diameter, divides into numerous large branches that end in contorted,
stout twigs. The leaf is twice pinnately compound, being made up of a hundred
or more separate oval leaflets arranged on branches of the rib. Its fruit
is a flat, leathery pod, 4 to 6 inches in length, containing 4 to 7 hard,
flat, dark brown seeds. Pods often remain on the tree until late winter.
The tree usually grows on rich bottom land and along streams, scattered
among other trees.
CONDITIONS OF POISONING. Poisoning is apt to occur in late winter
or early spring when other forage is scarce and animals eat quantities
of the pods and seeds lying on the ground, or nibble on young shoots. Cases
of poisoning from Kentucky coffee tree are not common, but they may be
fatal.
SYMPTOMS. These have not been well described, although, in reported
cases, animals seemed to show evidence of severe pain shortly after feeding
on leaves and pods. Profuse diarrhea with straining has also been reported.
At autopsy of these animals, a white, sticky mucus was present in the small
intestine with congestion of the mucous membranes, and foamy fecal matter
was found in the large intestine.
TREATMENT. There is no specific or reliable treatment. Veterinarians
may give symptomatic treatments that have proved valuable in other plant
alkaloid poisonings. However, recovery will depend largely on severity
of the poisoning.
Yellow sweet clover (Melilotus officinalis L. Lam)
Alkaloid. Plant substance, often poisonous, containing a basic
nitrogen grouping in its formula.
Alternate. Of leaves: placed singly at each joint, contrasting
with opposite and whorled arrangement.
Annual. Of plants: completing its growth in a single year or
season.
Asphyxia. Death due to deficiency of oxygen.
Astringent. Agent which causes the constriction of the tissues
by direct action.
Calyx. The outer, usually green and leaf-like part of a flower.
Capsule. A dry fruit that splits, at maturity, into 2 or more
valves.
Compound leaf. One which is divided into separate leaflets.
Dilated. Of eye: pupil widened or enlarged.
Frond. The "leaf" of a fern.
Glucoside. A plant substance, often poisonous, which by chemical
reaction with water yields a sugar.
Head. Type of dense flower cluster found in the composite family.
Herbaceous plant. One which develops no woody stem and is more
or less soft and green throughout.
Leaflet. One of the divisions of a compound leaf.
Opposite. Of leaves: two leaves at each stem joint.
Palmate. Of a compound leaf: having the leaflets radiating from
a common point.
Perennial. Of plants: continuing to live longer than two years.
Pinnate. Of a compound leaf: having the leaflets arranged on
each side of a common axis.
Saponin. A substance found in plants, often poisonous, which
is characterized by its property of producing a soapy lather.
Spore. The reproductive body in such plants as ferns and horsetails.
Sterile. Not producing seeds or spores.
Symptomatic. Of treatment: according to the symptoms of the
disease.
Terminal. Growing at the end of a branch or stem.
Toxic principle. The poisonous substance in a plant.
Umbel. Type of flower cluster in which the flowerstalks arise
from the same point like the ribs of an umbrella.
Whorl. Of leaves: having more than two leaves at each stem joint.